where is the set of all subjects is the

where is the set of all subjects, is the set of all raters, is the lesion mask from rater r, is the lesion mask for the algorithm A, and Corr is the Pearson׳s correlation coefficient of the volumes. This is then linearly normalized by the inter-rater scores between each rater such that the lower inter-rater score has an overall rating of 90. This was designed to mimic the scoring of the 2008 MICCAI MS Lesion challenge [8].
Acknowledgments This work was supported in part by the NIH/NINDS Grant R01-NS070906, by the Intramural Research Program of NINDS, and by the National MS Society Grant RG-1507-05243. Prizes for the challenge were furnished by the National MS Society.
Data The dataset is a collection of six multi-contrast melatonin receptor agonists MRI atlases, accompanied by the associated probabilistic maps for three main brain tissue types, segmented labels for 8 subcortical nuclei, and a co-registered histology-based atlas. Derived from 3T MRI scans of a cohort of 25 Parkinson׳s disease patients, the atlases were obtained by nonlinearly co-registering each patient׳s anatomy to a common space. The finished atlases are in MNI ICBM152 stereotactic space, with three image resolutions available: 1×1×1mm3, 0.5×0.5×0.5mm3, and 0.3×0.3×0.3mm3.
Experimental design, materials and methods
Acknowledgements This study was supported by Grants from CIHR (MOP-97820), NSERC (CHRPJ 385864) and FRSQ.
Data
Experimental design, materials and methods
Acknowledgements This work was made possible by Grants R21NS098018, R01HD065955, 2K24DA16170, U54NS056883, G12MD007601-26, and P41EB015909 from the National Institutes of Health, and Grant 46039500 from the Central Norway Regional Health Authority. The contents of this paper are solely the responsibility of the authors and do not necessarily represent the official view of NIH or the Central Norway Regional Health Authority. The authors are grateful to the families of our research participants, the pediatricians/neonatologists who referred the participants (Dr. Lillian Fujimoto, Dr. Lois Chiu, and Dr. Joseph Hudak), and our dedicated research staff (Steven Buchthal, Eric Cunningham, Daniel Alicata, Heather Johansen, Antonette Hernandez, Robyn Yamakawa, Sara Hayama, Tamara Andres), who assisted with the data collection. We also thank our board-certified neuroradiologist, Dr. Doris Lin, for her radiological reading of the scans. Lastly, we appreciate the instructive discussion with Dr. Laurent Younes about the development of mathematical model and statistical analysis.
Data Resveratrol hyposensitized breast cancer cells to various UVC irradiations (0–30mJ/sec) compared to cells treated with DMSO (Fig. 1). Next, resveratrol reduced more the S-phase cell cycle profiles post to 30mJ/sec of UVC-induced DNA damage, compared to the cells treated with UVC alone (See Fig. 2A and B). In addition, the effect of resveratrol on response of γ-H2AX induced by UVC treatment was provided in the western blotting (Fig. 2C).
Experimental design, materials and methods
Acknowledgments This study was supported by Basic Science Research Program through the National Research of Korea (NRF) funded by the Ministry of Science, ICT & Future Planning (Grant no. 2015R1C1A1A02037579) and Hannam University Research Fund (No. 2016).
Experimental design, materials and methods Two recent publications reported contradictory findings from analyses of data from the National Health and Nutrition Examination Survey (NHANES). Vozoris reported a statistically significantly increased adjusted odds of stroke diagnosis among menthol compared with non-menthol cigarette smokers, in particular among non-African Americans, using data from 2007–2008 cycle (incorrectly reported as 2001–2008) of NHANES [5]. Rostron did not detect a difference in stroke risk among smokers of menthol compared with non-menthol cigarettes, based on analyses of NHANES data from the 1999 through 2010 cycles [3]. Our investigation of the reasons for the discordant results reported by Vozoris and Rostron with respect to stroke risk, and the results of new analyses comparing stroke risks among smokers of menthol and non-menthol cigarettes that use all NHANES cycles from 1999 through 2012 is available elsewhere [4]. The differences between the Vozoris [5] and Rostron [3] results were shown to be mainly due to the inadvertent exclusion of all but the 2007–2008 NHANES data from the Vozoris [5] analysis. The data presented here examine risks of other endpoints evaluated by Vozoris (i.e., hypertension (HTN), myocardial infarction (MI), congestive heart failure (CHF), and chronic obstructive pulmonary disease(COPD)) among smokers of menthol compared with non-menthol cigarettes estimated according to three different logistic regression models: 1) models proposed by Vozoris, using NHANES 2007–2008, 1999–2010, and 1999–2012; 2) models proposed by Rostron, using NHANES 2007–2008, 1999–2010, and 1999–2012; and 3) a new set of models we developed with purposeful selection techniques using NHANES 1999–2012.